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Metabolic plasticity drives specific mechanisms of chemotherapy and targeted therapy resistance in metastatic colorectal cancer
8-isoprostane and leukotrienes were measured in EBC by enzyme immunoassay
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Consistent with the effects seen in rat PSCs, high glucose and TGFβ1 enhanced the proliferation and decreased the apoptosis of hPSCs under low glucose conditions, whereas GSH and SB431542 prevented these effects in the presence of high glucose (Fig
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