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The molecular evidence is genuinely compelling: Semax upregulates BDNF and NGF through clearly defined CREB-mediated pathways, modulates dopaminergic and serotonergic neurotransmission through sensitization rather than brute-force stimulation, suppresses neuroinflammatory cascades at the transcriptomic level during ischemic injury, and exerts immunomodulatory effects that extend to adaptive immunity
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