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Focusing on ferroptosis as a means to improve radiation therapy outcomes is a promising approach that warrants further exploration (Fig

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Introduction The key mechanism of ferroptosis involves the upregulation of lipid peroxidation pathways driven by iron, reactive oxygen species (ROS), and polyunsaturated fatty acids (PUFAs), coupled with the downregulation of antioxidant mechanisms, primarily selenium-dependent glutathione peroxidase 4 (GPX4) (1)