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Therapeutic treatment can encourage the replacement of damaged and lost neurons from available neural stem cells, encourage the formation of new functional synaptic connections among existing neurons, and facilitate cerebral blood flow and neuroprotection

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Signaling interactions between c-Met and the mutant epidermal growth factor receptor (EGFR) have been studied extensively, but signaling intermediates and biological consequences of lateral signaling to c-Met in EGFR wild-type tumors are minimally understood