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For example, mitochondrial dysfunction destabilizes lysosomes, amplifying oxidative stress ( 2.2.2 Endoplasmic reticulum: regulator of lipid metabolism and redox homeostasis The ER maintains cellular homeostasis through protein synthesis, lipid metabolism, and calcium storage ( - —localized to the ER membrane—mediates cystine import for GSH synthesis ( 2.2.3 Lysosomes: controllers of iron homeostasis Lysosomes regulate cellular debris degradation, biomolecule recycling, and iron metabolism—all of which influence ferroptosis ( 2.2.4 Lipid droplets: sources of polyunsaturated fatty acids Lipid droplets (LDs) are dynamic organelles serving as storage reservoirs for neutral lipids, playing pivotal roles in cellular energy homeostasis and lipid metabolism ( 2.2.5 Peroxisomes: modulators of lipid oxidation and antioxidant defense Peroxisomes regulate lipid metabolism and ROS detoxification, influencing ferroptosis (111)

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Cuprizone treatment impairs visual function, which was demonstrated by multifocal electroretinograms (mfERGs), an established non-invasive method, and Dock3 overexpression prevented this effect