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The role of platelets in immune-mediated inflammatory diseases | Nature Reviews Immunology
Thus, hTERT promotor mutation can be recognized as a novel biomarker and therapeutic target molecule for GBMs (184,186,187)
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[63] showed that PINK1 directly recruits optineurin (OPTN) and NDP52 to damaged mitochondria in a Parkin-independent manner and subsequently recruits and activates the autophagy initiator unc-51-like kinase 1 (ULK1), double FYVE-containing protein 1 (DFCP1) and WD repeat domain phosphoinositide interacting 1 (WIPI1), which in turn induce mitophagy (Figure 3)
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