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Ultimately, it participates in the initiation of immune inflammation, activation of cell death, and ATP-induced cell death, resulting in the release of inflammatory mediators such as IL-1β, IL-18 (interleukin-18), TNF-α, IL-2 (interleukin-2), IL-4 (interleukin-4), IL-6 (interleukin-6), IL-a (interleukin-A), CCL5 (chemokine (C-X-C motif) ligand 5), and CXCL2 (chemokine (C-X-C motif) ligand 2), ultimately culminating in cell death
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It cannot be excluded that callose may also have an impact on microspore embryogenesis (Parra-Vega et al., 2015) possibly having impact on GPRE
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