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In particular, prophylactic and prolonged glutathione infusion is able to reduce NOX2 activation and modulate inflammatory effector cell response as assessed by changes of differential leukocytes count and TNF‐α production over the first 5 days after STEMI
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However, GSH still caused a pronounced decrease in the NO peak height and an increase in the apparent decay rate in closed vessels and gave rise to sizeable NO signals after CuSO 4 addition (Fig
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