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The current 7 T MRS study has two primary objectives: first, to investigate neurometabolite levels in adults with ASD to elucidate its neurochemical pathophysiology, and second, to examine their association with symptoms of ASD
NMN (Nicotinamide Mononucleotide)
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Aspirin and sodium salicylate are proposed to inhibit transcription factor NF-κB-mediated pro-inflammatory signaling by inhibiting Iκκ-β kinase activity, which induces degradation of inhibitory protein of NF-κB (IκB) by phosphorylation (68, 69)
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